Human BH3-only protein, Noxa, is a major contributor to proliferative metabolism and apoptosis in the CD8 +T cell immune response

نویسندگان

چکیده

Abstract Human Noxa, the smallest BH3-only Bcl-2 protein, was originally identified as pro-apoptotic, but has since also been shown to promote cell growth in both malignant and activated normal T cells by facilitating proliferative metabolism (Lowman et al., 2010, Mol Cell, 40, 823–33; Hanse 2017, Oncogene, 36, 3915–3924). To investigate contribution of Noxa immune response, we tracked its expression primary human CD8 +T an vitro anti-CD3/CD28 co-stimulation model. is undetectable naïve induced upon remains highly expressed during differentiation phase through onset apoptosis. The viable that remain following apoptosis at end response are ‘memory-like’, determined flow cytometry, harboring large mitochondria with robust spare respiratory capacity. These no longer express rapidly upregulate it re-stimulation. We observe protein cells, stimulation, directly dependent on glutamine unaffected glutaminase inhibitors, while entry carbons into TCA cycle, turn, requires Noxa. NoxaKO moreover, show reduced dependence significantly higher viability response. expressing deficient BH3 domain point mutant similarly presented extended effector phase. fundamental contributions activation-associated metabolic reprogramming a prototypic makes this attractive tool manipulate expansion, function, longevity for immunotherapy. R21 AI148876

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ژورنال

عنوان ژورنال: Journal of Immunology

سال: 2023

ISSN: ['1550-6606', '0022-1767']

DOI: https://doi.org/10.4049/jimmunol.210.supp.226.08